Korean scientists can unravel how the mechanism that signals human hunger works. Will it be possible to deceive you?
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We eat when we are hungry.
We stop when we feel satiated.
And we don't think about what controls these sensations.
But one study revealed the mechanism behind the enzyme that regulates appetite.
The research is from the Daegu Gyeongbuk Institute of Science and Technology (South Korea).
It found that when we are low in glucose, the enzyme AMPK is activated.
This enzyme modifies the neuropeptides (protein molecules) that the brain uses to communicate.
Two of AMPK's components play a role in regulating hunger.
Together, these components help determine when we should start and stop eating.
To reach these conclusions, tests were performed with guinea pigs.
The animals were divided into two groups, fed the same diet.
Those with defective AMPK enzymes ate considerably less.
And also lost weight.
The discovery is expected to help create new drugs to combat obesity.
It adds to the arsenal of studies that seek a safe way to control weight gain.
Like a pill accidentally discovered by Yale University.
The new drug can let you eat as much as you want without getting fat.
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